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Averagebone-to-plasma AUC ratios for teicoplanin were 0.12 for cortical (n=17 patients) and0.56 for cancellous bone (n=15). One explanation offered for thediscrepancy between its analgesic-antipyretic andantiinflammatory actions is its inability to inhibitCOX in the presence of peroxides which aregenerated at sites of inflammation, but are notpresent in the brain. Increased level of ROS causes oxidative modification of proteins and nucleic acids andlipid peroxidation.
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